"Silicon is an essential element in biology, although only tiny traces of it appear to be required by animals.[8] However, various sea sponges as well as microorganisms like diatoms and radiolaria secrete skeletal structures made of silica. Silica is often deposited in plant tissues, such as in the bark and wood of Chrysobalanaceae and the silica cells and silicified trichomes of Cannabis sativa, horsetails and many grasses."
http://globalskywatch.com/chemtrails/ubbthreads.php?ubb=showflat&Number=14897#Post14897Dr. Hildegard Staninger
http://globalskywatch.com/chemtrails/ubbthreads.php?ubb=showflat&Number=15726#Post15726David Evered; Maeve O'Connor
Silicon Biochemistry
Chichester [England] ; New York : Wiley, 1986.
Ciba Foundation symposium, 121.
http://www.worldcat.org/oclc/13269865https://en.wikipedia.org/?title=NovartisEffects of Silica on Lung Collagen
Jerold A. Last andKaren M. Reiser
http://www.worldcat.org/oclc/13269865http://onlinelibrary.wiley.com/doi/10.1002/9780470513323.ch11/summary"Summary
A single intratracheal injection of 50 mg crystalline silica (quartz) into rats produces silicosis. This animal model may be used to study collagen metabolism during the early, middle, and late phases of lung injury, corresponding respectively to the stages of lung injury, development of discrete granulomas, and development of mature silicotic nodules. The early phase is characterized by a rapid increase in the rate of synthesis of lung collagen (within one week of instillation) and increased deposition of excess lung collagen (significant increases within two weeks of instillation). Later phases are characterized by a continuing increase in deposition of excess lung collagen for at least one year after instillation. Silica-induced fibrosis is unique among all the animal models (and in most human fibrotic diseases) thus far examined, in that the excess collagen deposited in the lung contains normal ratios of the two major collagen types of the lung: types I and III. This collagen is nonetheless biochemically different from normal lung collagen. There are reproducible and characteristic differences in the intermolecular cross-links of the collagen in lungs from rats injected with silica. Within one month of silica instillation (the earliest time point examined thus far), an increased hydroxylysine content of collagen can be appreciated. The reducible difunctional cross-links are also more likely to be derived from hydroxylysine (i.e. the ratio of dihydroxylated to monohydroxylated cross-links increases). Within four months of silica instillation (and increasingly thereafter), increased amounts of the mature trifunctional cross-link hydroxypyridinium (derived from three residues of hydroxylysine) can also be appreciated, seemingly paralleling the evolution of mature silicotic nodules in these lungs. These changes in cross-linking of lung collagen seem to be common to all the animal models of pulmonary fibrosis examined, and are also consistent with changes occurring in human fibrotic lungs. Preliminary observations suggest that the locus of cross-linking remains the same: hydroxylysine replaces lysine in the primary structure of a specific collagen α chain to form the altered cross-links. Thus, there may be molecular markers for the collagen of fibrosis in diseased lungs."
http://onlinelibrary.wiley.com/doi/10.1002/9780470513323.ch11/summaryEdwardson JA, Klinowski J, Oakley AE, Perry RH, Candy JM.
Aluminosilicates and the ageing brain: implications for the pathogenesis of Alzheimer's disease.
in: Ciba Found Symp. 1986;121:160-79.
http://www.ncbi.nlm.nih.gov/pubmed/3743229http://www.worldcat.org/oclc/13269865"Abstract
Senile plaques are a neuropathological feature of the ageing brain and consist of abnormal neuritic and glial processes surrounding an extracellular core of material with fibrillary ultrastructure. Present at low densities in the cerebral cortex of most aged individuals, they occur in large numbers in Alzheimer's disease, the major form of senile dementia. Energy-dispersive X-ray microprobe analysis of isolated cores and plaques in situ from patients with Alzheimer's disease or Down's syndrome and from normal controls has shown co-localization of high concentrations of aluminium (4-19%) and silicon (6-24%) at the centre of the core. The presence of these elements as aluminosilicates has been confirmed using solid-state 27Al nuclear magnetic resonance. These findings provide a link with the other major neuropathological feature of Alzheimer's disease, the neurofibrillary tangle-bearing neurons, where high intracellular levels of Al and Si have also been reported. The focal deposition of these elements may be an early and essential factor in the pathogenesis of Alzheimer-type changes, reflecting an increased exposure to aluminium."
http://www.ncbi.nlm.nih.gov/pubmed/3743229Silicon Biochemistry
Ciba Foundation symposium
John Wiley & Sons, 30.04.2008 - 272 Seiten
https://en.wikipedia.org/?title=NovartisSilicon is the second most abundant element in the Earth's crust, and is found in water, plants and organisms. The contributors decribe how silica gets into and out of organisms and discuss how essential or harmful silicon or silicon-based compounds are in higher animals.
longlink.com"Scientific sensation - Forbidden discovery of 1988"
http://globalskywatch.com/chemtrails/ubbthreads.php?ubb=showflat&Number=13015#Post13015https://en.wikipedia.org/?title=NovartisAntonio & Rosario Marcianò (Tanker Enemy)
http://globalskywatch.com/chemtrails/ubbthreads.php?ubb=showflat&Number=14897#Post14897http://onlinelibrary.wiley.com/doi/10.1002/9780470513323.ch7/summary
